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A novel acquired EGFR-SEPT14 fusion confers differential drug resistance to EGFR inhibitors in lung adenocarcinoma
Weidong Wang1,5,6,7; Wang Lv1; Hui Wang1; Yang Xu3; Junrong Yan3; Han-Ming Shen4,5; Liqun Shan1,2; Jian Hu1
2023-04-24
MediaGenes & Diseases
DOI10.1016/j.gendis.2023.02.019
Language英語English
URLView the original
Scopus ID2-s2.0-85162199986
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Document TypeOther
CollectionFaculty of Health Sciences
Corresponding AuthorLiqun Shan; Jian Hu
Affiliation1.Department of Thoracic Surgery,The First Affiliated Hospital,School of Medicine,Zhejiang University,Hangzhou,Zhejiang,310003,China
2.Department of Thoracic Surgery,The First People's Hospital of Wenling,Wenling,Zhejiang,317599,China
3.Medical Department,Nanjing Geneseeq Technology Inc.,Nanjing,210000,China
4.Department of Physiology,Yong Loo Lin School of Medicine,National University of Singapore,Singapore,119077,Singapore
5.Faculty of Health Sciences,University of Macau,Macau,519000,China
6.Department of Thoracic Surgery,Sun Yat-sen University Cancer Center,Guangzhou,Guangdong,510060,China
7.Sun Yat-sen University Cancer Center,State Key Laboratory of Oncology in South China,Collaborative Innovation Center for Cancer Medicine,Guangzhou,Guangdong,510060,China
First Author AffilicationFaculty of Health Sciences
Recommended Citation
GB/T 7714
Weidong Wang,Wang Lv,Hui Wang,et al. A novel acquired EGFR-SEPT14 fusion confers differential drug resistance to EGFR inhibitors in lung adenocarcinoma. 2023-04-24.
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